◀ Back to SLC22A3

KLF4 — SLC22A3

Text-mined interactions from Literome

Liu et al., Mol Cell Biol 2012 (Prostatic Neoplasms) : Expression of a degradation-resistant KLF4 mutant inhibited EMT, and furthermore, depletion of Klf4 was sufficient to initiate SLUG dependent EMT
Tiwari et al., PloS one 2013 (Neoplasms, Experimental) : Loss and gain of function experiments demonstrate that the down-regulation of Klf4 expression is required for the induction of EMT in vitro and for metastasis in vivo ... The data demonstrate a central role of Klf4 in the maintenance of epithelial cell differentiation and the prevention of EMT and metastasis