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FAS — PIK3R1
Pathways - manually collected, often from reviews:
Text-mined interactions from Literome
Chang et al., J Lipid Res 2005
:
In summary, we conclude that KGF requires both
PI3K and JNK signaling pathways to induce SREBP-1, which in turn
induces SCD-1 and
FAS expression in H292 cells
Kondo et al., Oncol Rep 2005
:
Although
PI 3-K inhibitors did not
affect Fas expression in HL-60 cells, cellular FLICE-inhibitory protein ( c-FLIP ) levels were markedly reduced by PI 3-K inhibitor treatment
Kondo et al., Oral Oncol 2006
(Carcinoma, Squamous Cell...) :
Although
PI 3-K inhibitors did not
affect the
Fas expression of OSCC cells, cellular FLICE-inhibitory protein ( c-FLIP ) levels were markedly reduced by PI 3-K inhibitor treatment
Chiang et al., Oncol Res 2005
(Carcinoma, Hepatocellular) :
FAS expression in HepG2 cells was strongly
inhibited by
PI3K inhibitor LY294002 and JNK inhibitor II and slightly inhibited by p38 inhibitor SB203580 and MEK inhibitor PD98059, separately
Bénéteau et al., Mol Cancer Res 2008
:
Herein, we show that inhibition of the
PI3K signal by edelfosine
triggers a
Fas mediated apoptotic signal independently of the Fas/FasL interaction
Zhang et al., Front Biosci (Elite Ed) 2011
:
Taken together, the present study demonstrates that T3 potentiates the cell survival action of FSH through Src- and
PI3K mediated Xiap up-regulation and decreased
Fas and FasL expression
Bhatt et al., Proc Natl Acad Sci U S A 2012
(Lymphoma, B-Cell...) :
We found that aerobic glycolysis and
FAS occur in a
PI3K dependent manner and appear to be interdependent