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CCL3 — IL13

Text-mined interactions from Literome

Waehre et al., J Am Coll Cardiol 2003 (Coronary Artery Disease) : Our main findings were : 1 ) gene expression of several chemokines ( i.e., macrophage inflammatory protein [MIP ] -1alpha, MIP-1beta, and interleukin [ IL]-8 ) and chemokine receptors ( i.e., CC chemokine receptor [ CCR]1, CCR2, CCR4, and CCR5 ) was markedly increased among CAD patients compared with healthy control subjects ; 2 ) treatment with atorvastatin and simvastatin markedly reduced the mRNA levels of some of these chemokines ( i.e., MIP-1alpha, MIP-1beta, IL-8 ) and receptors ( i.e., CCR1 and CCR2 ), with the most pronounced effect in the atorvastatin group ; and 3 ) statin therapy reduced the spontaneous release of IL-8 and MIP-1alpha from PBMCs in CAD patients
Ma et al., J Immunol 2004 (Inflammation) : C10/CCL6 neutralization also decreased the ability of IL-13 to stimulate the production of monocyte chemoattractant protein-1, macrophage inflammatory protein-1alpha , MMP-2, MMP-9, and cathepsins-K, -L, and -S and the ability of IL-13 to inhibit alpha1-antitrypsin
Wang et al., Arthritis Rheum 2013 (Intervertebral Disc Degeneration...) : Tumor necrosis factor a- and interleukin-1ß dependent induction of CCL3 expression by nucleus pulposus cells promotes macrophage migration through CCR1
de Waal Malefyt et al., J Immunol 1993 : Furthermore, IL-13 inhibited production of IL-1 alpha, IL-1 beta, IL-6, IL-8, IL-10, IL-12 p35, IL-12 p40, macrophage inflammatory protein-1 alpha , granulocyte/macrophage-CSF, granulocyte-CSF, IFN-alpha, and TNF alpha by monocytes activated with LPS
Berkman et al., Am J Respir Cell Mol Biol 1996 : Interleukin 13 inhibits macrophage inflammatory protein-1 alpha production from human alveolar macrophages and monocytes ... In AM, IL-13 reduced LPS induced MIP-1 alpha protein release of 2,030 +/- 242 pM by 32 +/- 8 % ( P < 0.05 ) and MIP-1 alpha mRNA by 27 +/- 1 % ( NS )
Minty et al., Eur Cytokine Netw 1997 : IL-13 does not significantly inhibit the IL-2 induced T lymphocyte production of IFN-gamma, RANTES, MIP-1 alpha or MIP-1 beta, nor that of perforin mRNA, as does IL-4