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IL1B — PTK7

Text-mined interactions from Literome

Lin et al., J Immunol 2000 : OX8 ( CD8 alpha ) -induced TNF and IL-1 beta production by macrophages was blocked by inhibitors of protein tyrosine kinase ( s ), PP1, and genistein, but not by phosphatidylinositol-3 kinase inhibitor, wortmannin
Chen et al., Mol Pharmacol 2000 : Taken together, IL-1beta activates phosphatidylcholine-specific phospholipase C and induces activation of PKCalpha and protein tyrosine kinase , resulting in the stimulation of NIK, IKK2, and NF-kappaB in the ICAM-1 promoter, then initiation of ICAM-1 expression
Lu et al., Chin Med J (Engl) 2000 (Arthritis, Rheumatoid) : Role of protein tyrosine kinase in IL-1 beta induced activation of mitogen activated protein kinase in fibroblast-like synoviocytes of rheumatoid arthritis
Raung et al., Biochem Biophys Res Commun 2005 (Encephalitis, Japanese) : PTK inhibitors suppressed JEV induced TNF-alpha and IL-1beta production at the transcriptional level
Shapira et al., J Immunol 1994 : Involvement of protein kinase C and protein tyrosine kinase in lipopolysaccharide induced TNF-alpha and IL-1 beta production by human monocytes ... The results suggest that TNF-alpha and IL-1 beta secretion after LPS stimulation of human monocytes requires the activation of protein tyrosine kinase and PKC, upstream to the activation of gene transcription
al-Daccak et al., Infect Immun 1994 (Arthritis, Infectious...) : Finally, our results, obtained with protein tyrosine kinase inhibitors and antiphosphotyrosine Western blotting ( immunoblotting ), indicate that protein tyrosine kinase is involved in MAM induced IL-1 beta and TNF-alpha gene expression in the THP-1 monocytic cell line
Trede et al., Clin Immunol Immunopathol 1994 : PTK and PKC activation plays a role in the induction of monokine gene transcription by SE because inhibitors of PTK and PKC reduced TSST-1 stimulated IL-1 beta and TNF-alpha gene expression
Simms et al., Ann Surg 1997 : Modulation of IL-1 beta and TNF-alpha receptors by VLA-5 and VLA-6 required protein tyrosine kinase activation as herbimycin A ( 10 micrograms/mL ) blocked the affect of Fn and Ln. Changes in PMN cytokine receptor expression led to parallel changes in functional activity as assessed by the binding of 125I IL-1 beta and TNF-alpha