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POLDIP2 — VCAM1

Text-mined interactions from Literome

Seye et al., J Biol Chem 2003 : The effect of UTP on VCAM-1 expression in HCAEC was prevented by depletion of intracellular calcium stores with thapsigargin or by inhibition of p38 mitogen activated protein kinase or Rho kinase, but was not affected by inhibitors of the mitogen activated protein/extracellular signal regulated kinase pathway ( i.e. MEK1/2 )
Wang et al., Am J Physiol Lung Cell Mol Physiol 2005 : Together, these results suggest that in HTSMC, activation of p42/p44 MAPK, p38 , JNK, and NF-kappaB pathways is essential for IL-1beta induced VCAM-1 gene expression
Lin et al., Cell Signal 2007 : Taken together, these results suggest that in HTSMCs, activation of p42/p44 MAPK, p38 , and JNK pathways, at least in part, mediated through NF-kappaB, is essential for LPS induced VCAM-1 gene expression
Madonna et al., Int J Immunopathol Pharmacol 2007 : Co-treatment of HUVEC with insulin and SB202190 strongly reverted the stimulatory effect of insulin on NF-KB activation, thus establishing a link between NF-KB activation and p38MAPkinase mediated induction of VCAM-1 by insulin
Hastings et al., Arterioscler Thromb Vasc Biol 2009 (Atherosclerosis) : Neutralization of the IL-8 receptor, CXCR2, further induced VCAM-1 in the presence of IL-1beta, and phospho-p38 was required for NF-kappaB activation and VCAM-1 expression
Hsu et al., J Cell Physiol 2011 : Furthermore, resistin increased the expression of intercellular adhesion molecule-1 ( ICAM-1 ) and vascular cell adhesion molecule-1 ( VCAM-1 ) by HUVECs and these effects were also p38MAPK dependent
Cho et al., The Korean journal of physiology & pharmacology : official journal of the Korean Physiological Society and the Korean Society of Pharmacology 2013 : SB202190, inhibitor of p38 mitogen activated protein kinase ( p38 MAPK ), and wortmannin, phosphatidylinositol 3-kinase ( PI3-kinase ) inhibitor, reduced LPS mediated VCAM-1 but not ICAM-1 expression