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LIF — STAT1
Text-mined interactions from Literome
Zhang et al., Exp Eye Res 2005
:
Retinas from embryonic and neonatal stages showed that STAT3 but not
STAT1 was activated in
response to ciliary neurotrophic factor (CNTF),
leukemia inhibitory factor (LIF) , fibroblast growth factor-1 (FGF1), fibroblast growth factor-2 (FGF2), epidermal growth factor (EGF), interferon-alpha ( IFN-alpha ) and interferon-gamma (IFN-gamma) in distinct patterns
Dekanty et al., J Biol Chem 2006
:
LIF and OSM specifically
trigger STAT1 cytoplasmic to nuclear translocation, whereas PGF2alpha fails to do so
Kurdi et al., Int J Biochem Cell Biol 2012
(MAP Kinase Signaling System) :
Unexpectedly,
LIF induced
STAT1 activation was unaffected by GSH depletion
Fahmi et al., Cell Signal 2013
(Hypertrophy...) :
In the absence of sIL-6R, IL-6 did not
induce STAT1/3 phosphorylation, whereas IL-6/sIL-6R and
LIF induced STAT1 and STAT3 phosphorylation
Ray et al., J Clin Invest 1996
(Pituitary Neoplasms) :
1 nM
LIF rapidly
induced tyrosyl phosphorylation of
STAT 1 and STAT 3 proteins, as well as tyrosyl phosphorylation of a 115-kD protein, coimmunoprecipitated with STAT 1