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EPHB2 — NOS1

Text-mined interactions from Literome

Chan et al., Infect Immun 2001 : Induction of inducible nitric oxide synthase-NO* by lipoarabinomannan of Mycobacterium tuberculosis is mediated by MEK1-ERK , MKK7-JNK, and NF-kappaB signaling pathways
Slomiany et al., Biochem Biophys Res Commun 2002 (MAP Kinase Signaling System) : Porphyromonas gingivalis lipopolysaccharide interferes with salivary mucin synthesis through inducible nitric oxide synthase activation by ERK and p38 kinase
Ebrahimian et al., J Hypertens 2003 : The ERK1/2 kinase cascade inhibitor PD 98059 significantly impaired the expression of nNOS in arteries exposed to 200 mmHg for 9 h
Raines et al., Biochem J 2006 : Previous studies from our laboratory have demonstrated that nNOS production of NO* blocks Ca2+-ionophore induced activation of ERK1/2 ( extracellular-signal regulated kinase 1/2 ) of the mitogen activated protein kinases through a mechanism involving Ras G-proteins and Raf-1 kinase
Choi et al., Eur J Pharmacol 2007 : Isoeugenol suppression of inducible nitric oxide synthase expression is mediated by down-regulation of NF-kappaB, ERK1/2 , and p38 kinase
Woo et al., Circ Res 2008 (Diabetes Mellitus...) : Shear stress induced extracellular signal regulated kinase ( ERK ) 5 activation and the consequent regulation of Kruppel-like factor 2 and endothelial nitric oxide synthase expression represents one of the antiinflammatory and vascular tone regulatory mechanisms maintaining normal endothelial function
Maulik et al., Neurosci Lett 2008 (Anoxia...) : Since a NOS inhibitor prevented the hypoxia induced phosphorylation of p38, ERK and JNK, we conclude that the hypoxia induced activation of p38, ERK and JNK in the cerebral cortical nuclei of guinea pig fetus at term is NO-mediated
Milkiewicz et al., J Cell Physiol 2008 : This response was modulated by ERK1/2 and p38 MAP kinases, but was not dependent on NOS or COX-2 activity
Shi et al., Nan Fang Yi Ke Da Xue Xue Bao 2009 (Parkinson Disease, Secondary) : [ Effect of phosphorylated-ERK1/2 on inducible nitric oxide synthase expression in the substantia nigra of mice with MPTP induced Parkinson disease ]
Surachetpong et al., PLoS Pathog 2009 (MAP Kinase Signaling System) : At the highest treatment dose of TGF-beta1, inhibition of ERK phosphorylation increased TGF-beta1 induced expression of the anti-parasite effector gene nitric oxide synthase (NOS) , suggesting that increasing levels of ERK activation negatively feed back on induced NOS expression
Lin et al., Chem Biol Interact 2009 : LPS- and LTA induced NOS/NO productions were significantly suppressed by the JNK inhibitor, SP600125, but not by the ERK inhibitor, PD98059, through a reduction in JNK protein phosphorylation
Ralay Ranaivo et al., Brain Res 2010 (MAP Kinase Signaling System) : Albumin activated ERK1/2 , p38 MAPK and JNK signaling pathways in astrocytes, and induced the production of interleukin (IL)-1beta, inducible nitric oxide (NO) synthase , the NO metabolite nitrite, and the chemokine CX3CL1 while reducing the level of S100B
Gallo et al., J Neurosci 2011 : In neuronal cultures, NOS inhibition attenuated the bicuculline induced activation of ERK and the expression of c-Fos, Egr-1, Arc, and brain derived neurotrophic factor (BDNF), proteins essential for neuroplasticity
Huang et al., J Biol Chem 2012 : Inhibition of ERK reduced both nNOS activation and NO secretion
Pignataro et al., Neurobiol Dis 2013 (Infarction, Middle Cerebral Artery) : In fact, p-ERK expression increased in brain cortex after remote postconditioning, and its pharmacological inhibition prevented both nNOS overexpression and remote postconditioning mediated neuroprotection