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CAMK1 — CREB3

Text-mined interactions from Literome

Hansen et al., J Neurosci Res 2003 (Calcium Signaling) : First, that depolarization of SGNs activates CaMKs is evidenced by observation of increased CaMKII phosphorylation and of CaMK dependent CREB phosphorylation
Ahi et al., Behav Brain Res 2004 : Inhibition of CaMK II did not affect fear conditioning and reduced the phosphorylation of Erk-1/2, Elk-1, CREB , and p90Rsk-1 only transiently, whereas Mek-1/2 inhibition was ineffective in all experiments
Egan et al., Cell Signal 2004 : In contrast, CREB activation induced by depolarization in both neonatal and developed VSM was significantly reduced by CaMK inhibition and by ERK1/2 inhibition
Mathieson et al., Br J Pharmacol 2006 : CREB activation by SPC required both p38MAPK and CaMK activation, but not ERK1/2
Francis et al., Am J Physiol Cell Physiol 2008 : In several cells : 1 ) activation of HRH1 increases intracellular Ca ( 2+ ) concentration levels ; and 2 ) increased [ Ca ( 2+ ) ] ( i ) levels are coupled with calmodulin dependent stimulation of calmodulin dependent protein kinase ( CaMK ) and activation of cAMP-response element binding protein ( CREB ) ... We measured IP ( 3 ), Ca ( 2+ ) and cAMP levels, phosphorylation of CaMK I , and activation of CREB ( in the absence/presence of W7 ) in small cholangiocytes treated with HTMT dimaleate
Sasaki et al., Neuron 2011 : These findings suggest that SIK2 plays critical roles in neuronal survival, is modulated by CaMK I/IV , and regulates CREB via TORC1
Houglum et al., J Clin Invest 1997 : Moreover, the phosphorylation of CREB on Ser133 was stimulated in activated cells by inducing the activity of PKA or CAMK-II