◀ Back to CAV1

CAV1 — CD36

Text-mined interactions from Literome

Frank et al., Biochemistry 2002 : On the other hand, caveolin-1 has little effect on SR-BI stability, but clearly increases CD36 stability
Frank et al., Arterioscler Thromb Vasc Biol 2004 (Aortic Diseases...) : Mechanistically, we demonstrated that loss of Cav-1 resulted in the dramatic downregulation of certain proatherogenic molecules, namely, CD36 and vascular cell adhesion molecule-1
Ring et al., Biochim Biophys Acta 2006 : In conclusion, our data provide evidence that caveolin-1 is necessary to target FAT/CD36 to the plasma membrane
Li et al., Atherosclerosis 2013 : The specific CD36 signal induced by oxLig-1 initiated the activation of Fyn, Lyn, caveolin-1 , JNK2 and ERK1/2, and resulted in the up-regulation of ABCA1