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JUN — SELE

Pathways - manually collected, often from reviews:

• NCI Pathway Database ATF-2 transcription factor network: JUN/ATF2 complex (JUN-ATF2) → ELAM1 (SELE) (transcription, activates) Read et al., J Biol Chem 1997*
  Evidence: physical interaction

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Innatedb Interaction: Complex of SELE-MAPK9-JUN (unknown, -) Read et al., J Biol Chem 1997*
• IRef Innatedb Interaction: SELE — JUN (unknown, -) Read et al., J Biol Chem 1997*
• IRef Innatedb Interaction: Complex of JUN-SELE-MAPK8 (unknown, -) Read et al., J Biol Chem 1997*

Text-mined interactions from Literome

Chiu et al., Blood 2007 : By using specific inhibitors, dominant negative mutants, and small interfering RNA, we demonstrated that activations of c-Jun-NH ( 2 ) -terminal kinase ( JNK ) and p38 of the MAPK pathways are critical for the coculture induced E-selectin expression
Ishizuka et al., Clin Exp Immunol 1998 : These findings suggest that ICAM-1 or ELAM-1 expression of HUVEC stimulated via TXA2 receptors is augmented by induction of NF-kappaB and AP-1 binding activity through the PKC system, and that VCAM-1 expression is augmented by induction of NF-kappaB binding activity