Gene interactions and pathways from curated databases and text-mining

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TGFB3 — TGFBR1

Pathways - manually collected, often from reviews:

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Franchi et al., Applied immunohistochemistry & molecular morphology : AIMM / official publication of the Society for Applied Immunohistochemistry 2001 (Bone Neoplasms...) : Transforming growth factor-beta2 and TGF-beta3 were detected in the majority of cases ( 97.1 % and 82.8 %, respectively ), whereas TGF-beta receptor type I ( TGF-beta RI ) and type II ( TGF-beta RII ) were diffusely expressed in all cases
Matsuzaki et al., Hepatology 2007 (Carcinoma, Hepatocellular...) : Conversely, transforming growth factor beta ( TGF-beta ) activates not only TGF-beta type I receptor ( TbetaRI ) but also c-Jun N-terminal kinase (JNK), which convert the mediator Smad3 into two distinctive phosphoisoforms : C-terminally phosphorylated Smad3 ( pSmad3C ) and linker phosphorylated Smad3 ( pSmad3L )
Alfranca et al., Blood 2008 : MT1-MMP dependent transforming growth factor beta ( TGFbeta ) signaling through Alk5 is also required for PGE ( 2 ) -induced endothelial cord formation in vitro, and Alk5 kinase activity is required for PGE ( 2 ) -induced neovascularization in vivo