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HEY1 — NOTCH2

Text-mined interactions from Literome

Takizawa et al., Nucleic Acids Res 2003 : In this paper, we show that BMP2 enhances Notch induced transcriptional activation of Hes-5 and Hesr-1 in mouse neuroepithelial cells
Kokubo et al., Dev Biol 2005 : Mouse hesr1 and hesr2 genes are redundantly required to mediate Notch signaling in the developing cardiovascular system
Ross et al., Mol Endocrinol 2006 : HRT1 , another basic-helix-loop-helix protein and potential Hes-1 partner, was also induced by Notch in 3T3-L1 cells but did not block adipogenesis, suggesting that Hes-1 functions primarily as a homodimer or possibly as a heterodimer with an unknown partner
Ader et al., Mech Dev 2006 : In addition, TGFbeta/BMP signaling pathway members including the type I TGFbeta receptor and Smads 3, 4 and 5 were significantly up regulated, as were several TGFbeta/BMP responsive genes including Hey 1 , a regulator of Notch pathway signaling
Li et al., Microvasc Res 2006 (Retinal Neovascularization) : HESR1 is a downstream target of Notch in endothelial cells and could be an effector of Notch signaling in these cells
Kokubo et al., Development 2007 : Furthermore, we find that the expression of Hesr1 and Hesr2 is independent of Notch2 signaling
Hu et al., Immunity 2008 : TLR- and Notch induced Hes1 and Hey1 attenuated IL-6 and IL-12 production
Keuylian et al., J Biol Chem 2012 (Carotid Artery Injuries...) : Conversely, Notch activation resulted in blocking AC8 expression and up-regulated Hrt1 and Hrt3 expression