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CRH — FOS

Text-mined interactions from Literome

Bittencourt et al., J Neurosci 2000 : Dual labeling revealed extensive correspondence of CRF stimulated Fos-immunoreactivity (Fos-ir) and CRF-R1 mRNA at these and other loci
Wang et al., Brain Res 2000 : Neither CRF nor urocortin induced Fos expression in the lateral septal nucleus, Edinger-Westphal nucleus, dorsal raphe nucleus, locus coeruleus, or hypoglossal nucleus
Miampamba et al., Am J Physiol Gastrointest Liver Physiol 2002 : The CRF ( 1 ) /CRF ( 2 ) antagonist astressin ( 33 microg/kg ip ) and the selective CRF ( 1 ) antagonist CP-154,526 ( 20 mg/kg sc ) prevented intraperitoneal CRF induced Fos expression in the proximal colon ( number of Fos-IR cells/ganglion : 2.7 +/- 1.2 and 1.0 +/- 1.0, respectively ), whereas atropine ( 1 mg/kg sc ) had no effect
Maillot et al., Brain Res 2003 : Intraperitoneal corticotropin releasing factor and urocortin induce Fos expression in brain and spinal autonomic nuclei and long lasting stimulation of colonic motility in rats ... CRF injected intraperitoneally ( i.p. ) stimulates colonic motor function and induces Fos expression in colonic myenteric neurons ... CRF increased significantly Fos expression in the central nucleus of the amygdala ( lateral part ), parabrachial nucleus ( external lateral subnucleus ), area postrema, nucleus tractus solitarius, locus coeruleus, paraventricular nucleus of the hypothalamus, the intermediolateral column and area I-VII, X at the L6-S1 level of the spinal cord by 11-, 6.5-, 5.3-, 5.0-, 4.7-, 2.7- and 1.4-fold, respectively compared with i.p. CRF ( 9-33 ) injected rats that had little Fos expression
Honkaniemi et al., Brain Res 1992 : Subsequently, Fos may regulate the expression of the NT, ENK, SOM and CRF genes and thus affect the peptidergic efferents from the ACe
Gammie et al., Behav Neurosci 2004 : Maternal aggression and icv CRF ( 0.2 microg ) induced Fos in 11 of the same regions, including the lateral and medial septum, the bed nucleus of the stria terminalis, the medial and central amygdala, the periaqueductal gray, the dorsal raphe, and the locus coeruleus
Wotus et al., Am J Physiol Regul Integr Comp Physiol 2007 : In WR rats, Fos increased in VP magnocellular and parvocellular neurons but not CRH neurons
Nunez et al., J Neurochem 2007 (Morphine Dependence) : To reveal secretory, activational and transcriptional changes in the hypothalamus of morphine dependent rats during naloxone precipitated opioid withdrawal, we measured corticosterone secretion, c-Fos induction and heteronuclear ( hn ) RNA levels of corticotropin releasing hormone (CRH) and arginine vasopressin (AVP) in naïve and morphine dependent animals injected with saline or 5 mg/kg naloxone
Wu et al., Am J Physiol Endocrinol Metab 2007 : CRH significantly increased mRNA and protein expression of c-Fos and Cx43 in a dose dependent manner ( P < 0.01 ). A transient transfection of AP-1 decoy ODN did not affect CRH regulation of c-Fos ( P > 0.05 ) but almost completely abolished CRH induced enhancement of Cx43 expression ( P < 0.01 )
Skórzewska et al., Horm Behav 2008 : Moreover, exogenous CRF increased aversive context induced expression of c-Fos in the parvocellular neurons of the paraventricular hypothalamic nucleus ( pPVN ), CA1 area of the hippocampus, and M1 area of the frontal cortex
Usui et al., Auton Neurosci 2009 : CRF significantly increased c-Fos expression in the celiac and stellate ganglia, with more pronounced effect on the celiac ganglion
Yamaguchi et al., Auton Neurosci 2009 : Therefore, in the present study, we investigated the effect of intracerebroventricularly ( i.c.v. ) administered CRF on plasma catecholamine levels and expression of NOS isozymes ( iNOS and nNOS ) and Fos ( a marker for neuronal activation ) in the spinally projecting PVN neurons, using rats microinjected with a monosynaptic retrograde tracer into the IML
Tan et al., Behav Brain Res 2009 : Teneurin C-terminal associated peptide (TCAP)-1 attenuates corticotropin releasing factor (CRF) induced c-Fos expression in the limbic system and modulates anxiety behavior in male Wistar rats ... Other areas, such as the paraventricular nucleus of the hypothalamus, bed nucleus of the stria terminalis, medial nucleus of the amygdala, and locus coeruleus, displayed CRF induced c-Fos levels that were unaffected by TCAP-1
Li et al., J Neuroendocrinol 2011 : CRF ( 25, 50 or 100 pmol in 200 nl of artificial cerebrospinal fluid ) administered bilaterally into the BNST resulted in a dose dependent decrease in LH pulse frequency, and induced Fos expression in glutamic acid decarboxylase immunostained neurones in the medial preoptic area
Gourcerol et al., Gastroenterology 2011 (Acute Disease...) : In rats, a selective agonist of CRF ( 2 ) ( urocortin 2 ) reduced CRF induced defecation ( > 50 % ), colonic contractile activity, and Fos expression in the colonic LMMP
Autelitano et al., J Neuroendocrinol 1994 (Pituitary Neoplasms) : Although DEX treatment of AtT-20 cells did not affect AP-1 DNA binding capacity of nuclear extracts, DEX pretreatment blunted the stimulation of AP-1 binding in response to CRF ... High levels of DEX treated extracts led to a relative repression of CRF induced AP-1 binding, suggesting that ligand activated GR may lower available AP-1 levels by direct protein : protein interaction ... These data suggest that in the corticotrope cell relatively high levels of activated GR may influence CRF induced AP-1 DNA binding via transient genomic actions on basal c-jun and stimulated c-fos and/or via direct protein : protein interactions
Rassnick et al., Neuroscience 1998 : A few brain regions, most notably the hypothalamic paraventricular nucleus, expressed Fos in response to foot shock but not corticotropin releasing hormone