◀ Back to AKT3

AKT3 — TGFB2

Text-mined interactions from Literome

Higaki et al., Arterioscler Thromb Vasc Biol 1999 : We also found that transforming growth factor-beta stimulated PI3K activity and the phosphorylation of Akt , a downstream signaling molecule of PI3K
Samatar et al., J Biol Chem 2002 : Transforming growth factor-beta 2 is a transcriptional target for Akt/protein kinase B via forkhead transcription factor
Yao et al., Ophthalmic Res 2008 : TGF-beta2 activated PI3K/Akt in a time dependent manner, but not extracellular signal regulated kinase and p38 MAPK
Wang et al., Mol Cell Biol 2008 (Breast Neoplasms) : In HER2 overexpressing mammary epithelial cells, transforming growth factor beta ( TGF-beta ) activated phosphatidylinositol-3 kinase (PI3K)/Akt and enhanced survival and migration
Caron et al., Reproductive biology and endocrinology : RB&E 2009 : Noteworthy, TGF-beta2 and TGF-beta3 reduced the level of the anti-apoptotic XIAP protein, as well as the level of phosphorylated/active Akt , a well known survival protein, in a dose dependent manner