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BCL10 — IRF6
Text-mined interactions from Literome
Fukui et al., J Immunol 2003
:
LPS stimulation
induced the expression of Fas, caspase 8, cellular FLIP Bfl-1/A1, and
Bcl-x , but not FasL, TNFR p55, Bak, Bax, and Bad at the transcriptional level
Liu et al., J Biol Chem 2004
:
In
response to
lipopolysaccharide (LPS) stimulation,
BCL10 was recruited to TLR4 signaling complexes and associated with Pellino2, an essential component down-stream of BCL10 in the TLR4 pathway ... In
response to
lipopolysaccharide (LPS) stimulation,
BCL10 was recruited to TLR4 signaling complexes and associated with Pellino2, an essential component down-stream of BCL10 in the TLR4 pathway
Sano et al., Res Vet Sci 2005
:
By real-time quantitative PCR analysis, it was indicated that
Bcl-xL and Bax levels in canine neutrophils were significantly
affected by
LPS stimulation
Yu et al., FASEB J 2005
(Inflammatory Bowel Diseases) :
Western blot showed that
LPS in high glucose
increased the levels of anti-apoptotic Bcl-2 and
Bcl-X ( L, ) and did not change proapoptotic Bax
Ramana et al., Free Radic Biol Med 2007
:
In addition, AR inhibition prevented the
LPS induced down-regulation of
Bcl-xl and up-regulation of Bax and Bak in macrophages
Bhattacharyya et al., Am J Physiol Gastrointest Liver Physiol 2007
(Inflammation) :
Bcl10 mediates
LPS induced activation of NF-kappaB and IL-8 in human intestinal epithelial cells
Lee et al., J Pineal Res 2009
(Body Weight...) :
In western blot analysis,
LPS reduced the levels of
Bcl-XL and procaspase-3 in SD mice