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GIT1 — INS

Text-mined interactions from Literome

Ostenson et al., J Clin Endocrinol Metab 1988 (Diabetes Mellitus, Type 2) : In the diabetic group iv administration of idazoxan 20 min before the GIT did not alter the insulin response to the GIT
Jurcovicová et al., Endocrinol Exp 1977 : The important role of GIT in insulin degradation seems to be conclusively demonstrated
Brush et al., Endocrinology 1979 : From these studies it was seen that 1 ) insulin is reduced at a very significant rate nonenzymatically ; 2 ) during short periods of incubation ( 30 sec ) where no significant hormone is reduced nonenzymatically, the rate of cleavage by the insulin protease present in the cytosol is extremely high and the microsomal GIT activity is negligible ; and 3 ) insulin destruction noted in isolated liver cells and perfused liver is most probably due to the insulin protease activity of the cytosol
Wajngot et al., Acta Endocrinol (Copenh) 1983 (Body Weight) : The insulin response to GIT was analysed by parameter identification in a mathematical model ( parameter KI )
Efendić et al., Horm Metab Res 1984 (Myocardial Infarction) : The post-coronary patients were not taking any drugs except for furosamide, which was shown not to influence insulin response to GIT or glucose tolerance
Karlsson et al., Can J Cardiol 1996 : To study how muscle ubiquinone content ( MUQ ) and percentage slow twitch ( % ST ) of muscle fibres in the thighs of male healthy subjects with a low or high insulin response to glucose infusion tests ( GIT ) affected blood glucose control ( BGC )
Liu et al., Amino Acids 1998 : ( ii ) in the cases when hepatic cationic amino acid transport is needed, such as following feeding, cellular growth and illness, glucocorticoids and insulin induce expression of the Cat-1 gene in liver cells through induction of transcription and stabilization of the mRNA