◀ Back to MSH4

MITF — MSH4

Text-mined interactions from Literome

Huber et al., J Biol Chem 2003 (Neuroblastoma) : Although M-MITF promoter activation by MSH is known to occur through a conserved cAMP-response element ( CRE ), it remains unclear how this CRE exhibits such exquisitely tissue restricted responsiveness
Kim et al., Biol Pharm Bull 2007 (Melanoma, Experimental) : Moreover, alpha-melanocyte stimulating hormone ( alpha-MSH ) is known to increase melanin biosynthesis by increasing tyrosinase production, and here, we found that alpha-MSH induced Mitf and tyrosinase increases were inhibited in B16 melanoma cells treated with KHG22394
Yang et al., Mol Cell 2008 : The activation of p53-POMC/MSH-MC1R signaling is required for the UV-induced melanogenic response because PAX3 functions in synergy with SOX10 in a cAMP-response element ( CRE ) -dependent manner to regulate the transcription of Mitf
Shoag et al., Mol Cell 2013 (Genetic Predisposition to Disease...) : Inhibition of PGC-1a and PGC-1ß blocks the a-MSH mediated induction of MITF and melanogenic genes