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ATP5O — PI3
Text-mined interactions from Literome
Alexander et al., Am J Physiol Gastrointest Liver Physiol 2001
:
Involvement of
PI 3-kinase in IGF-I stimulation of jejunal
Na+-K+-ATPase activity and nutrient absorption
Muscella et al., J Cell Physiol 2005
(Breast Neoplasms) :
Additionally, the effect of Ang II on
Na+/K+ATPase activity was also
blocked by the
phosphatidylinositol 3-kinase (PI3K) inhibitors, wortmannin and LY294002, and by the actin depolymerizing agents, cytochalasin D
Garofalo et al., Proc Biol Sci 2009
:
Using the same bioassay, we now demonstrate that this effect is confirmed in the presence of the exogenous NO donor S-nitroso-N-acetyl penicillamine, is independent from endocardial endothelium and guanylate cyclase/cGMP/protein kinase G and cAMP/protein kinase A pathways, involves a
PI(3)kinase mediated activation of endothelial NO synthase and a modulation of the
SR-CA(2+)ATPase ( SERCA2a ) pumps
Lei et al., Am J Physiol Lung Cell Mol Physiol 2011
:
Inhibitors of Src kinase ( PP1 ),
PI3K ( wortmannin ), and ERK1/2 ( U0126 ) all
blocked the T3-induced
Na-K-ATPase activity