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Text-mined interactions from Literome

Saeki et al., Biochem J 2002 (MAP Kinase Signaling System) : In addition, dithiothreitol also suppressed both apoptosis and JNK/p38 activation by EGCG, and EGCG induced activation of MAP kinase kinase (MKK) 3/6, MKK4 and apoptosis regulating kinase 1 (ASK1) was suppressed by NAC
Kyaw et al., J Pharmacol Sci 2004 : We found that L-NAC and D-NAC both inhibited Ang II-induced c-Jun N-terminal kinase and p38 mitogen activated protein kinase activation and [ ( 3 ) H ] -thymidine incorporation in VSMC
Lin et al., J Neurosci Res 2006 : Farnesyltransferase and MEK inhibitors completely abolished NAC induced p38 phosphorylation whereas p38 inhibitor did not influence NAC induced ERK phosphorylation
Shastry et al., Kidney Int 2007 : Hcy increased p38-MAPK activity ( fivefold ), with maximal effect at 50 microM and 20 min ; p38-MAPK activation was attenuated by N-acetylcysteine (Nac) and catalase (Cat), further indicating that the effect was via oxidative stress
Zhang et al., Toxicological sciences : an official journal of the Society of Toxicology 2011 : Moreover, NAC significantly attenuated c-Jun NH2-terminal kinase and p38 mitogen activated protein kinase phosphorylation induced by TGHQ
Krifka et al., Biomaterials 2012 : The antioxidant N-acetylcysteine (NAC) protected cells from TEGDMA induced cell death, and inhibited the activation of ERK1/2, p38 and JNK by TEGDMA