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NA — PVR

Text-mined interactions from Literome

Lei et al., Am J Pathol 2010 (Disease Models, Animal...) : Finally, these observations with an animal model appear relevant to clinical PVR because NAC prevented human PVR vitreous induced contraction of primary RPE cells derived from a human PVR membrane
Ardolino et al., Blood 2011 : Oxidative stress contributed to DDR activation, and our results showed impaired PVR levels in the presence of the reactive oxygen species ( ROS ) scavenger N-acetyl-cysteine (NAC) , being monocytes the main ROS source needed for optimal PVR expression on activated T cells