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AVP — NOS2

Text-mined interactions from Literome

Fan et al., Life Sci 2007 (Cardiomyopathies...) : AVP also increased NOS activity, protein and mRNA levels of iNOS in a coincident manner ... In addition, it was found that NF-kappaB was activated by AVP , and that PDTC could inhibit NO production, NOS activity, protein and mRNA levels of iNOS stimulated by AVP in a dose dependent manner
Fan et al., Zhongguo Ying Yong Sheng Li Xue Za Zhi 2007 : AVP significantly increased iNOS mRNA expressions, NOS activity and NO contents ( P < 0.05 ) in CFs. IL-1beta enhanced the effects of AVP on iNOS-NO system activity in a concentration dependent manner, moreover the iNOS mRNA expressions, NOS activity and NO contents of AVP + 3 ng/ml, AVP + 5 ng/ml IL-1beta group were both significantly higher than those of AVP group ( P < 0.05 )
Yamamoto et al., Hypertens Res 1997 : These results indicate that AVP inhibits IL-1 beta induced iNOS expression in VSMCs through the V1a receptor, which is mediated at least partially via activation of protein kinase C
Yamamoto et al., Hypertension 1997 : We investigated the effects of arginine vasopressin (AVP) on nitric oxide ( NO ) synthase activity in cardiac myocytes by measuring the production of nitrite, a stable metabolite of NO, and the expression of inducible NO synthase (iNOS) mRNA and protein
Raber et al., Brain research. Brain research reviews 1998 (Disease Models, Animal...) : The IFNalpha stimulated release of AVP and CRH appears to be mediated by cyclic GMP production, and this release by IFNalpha and IL-2 may be mediated in part by activation of constitutive nitric oxide synthase