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PRKAR2B — RNF19A

Text-mined interactions from Literome

Park et al., Neurosci Lett 2000 : Here we demonstrate that PKA is required for depolarization induced activation of both extracellular signal regulated kinase ( ERK ) and p38 mitogen activated protein ( MAP ) kinase in PC12 cells
Schulte et al., Exp Cell Res 2003 : Inhibition of cAMP dependent protein kinase (PKA) using H89 ( N- [ 2- ( ( p-bromocinnamyl ) amino ) ethyl ] -5-isoquinolinesulfonamide ) ) blocked phosphorylation of the cAMP response element binding protein ( CREB ) and p38 , but did not decrease NECA induced ERK1/2 phosphorylation
Hildesheim et al., J Biol Chem 2005 (Adenomatous Polyposis Coli...) : Moreover, lack of normal p38 MAPK activity in either Gadd45a-null keratinocytes or in p38 MAPK inhibitor treated keratinocytes leads to decreased CK2 activity and increased PKA activity
Chen et al., Cancer Res 2007 (Breast Neoplasms) : Taken together, PGE ( 2 ) activation of p38 and JNK1 via PKA and PKC is necessary for aromatase induction in BAFs, and p38 and JNK1 are potential new drug targets for tissue-specific ablation of aromatase expression in breast cancer
McAlees et al., Mol Cell Biol 2009 : beta ( 2 ) AR agonists, cAMP elevating agents, and PKA inhibitors were used to show that beta ( 2 ) AR stimulation resulted in a PKA dependent increase in p38 MAPK phosphorylation