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ANGPT2 — CREB5

Text-mined interactions from Literome

Reddy et al., American journal of physiology. Renal physiology 2002 (Diabetic Nephropathies...) : ANG II and 12 ( S ) -HETE led to activation of p38 ( MAPK ) and its target transcription factor cAMP-responsive element binding protein ( CREB )
Kang et al., J Mol Endocrinol 2006 (MAP Kinase Signaling System) : Additionally, Ang-II enhanced the DNA binding activity to cAMP response element binding protein ( CREB ) and phosphorylation of CREB
Fukuyama et al., Arterioscler Thromb Vasc Biol 2006 (Hyperthyroidism) : 3,3',5-triiodo-L-thyronine ( T3 ) did not show a significant effect on Ang II-induced activation of extracellular signal regulated protein kinase or p38 mitogen activated protein kinase in vascular smooth muscle cells ( VSMCs ), whereas T3 inhibited Ang II-induced activation of cAMP response element ( CRE ) binding protein ( CREB ), a nuclear transcription factor involved in the vascular remodeling process
Tsai et al., Circ Res 2007 : In the rat model, Ang II infusion increased LCC alpha1C subunit expression and serine 133 phosphorylation of CREB , which were attenuated by oral losartan and simvastatin
Pham et al., J Cell Physiol 2008 : Ang II and EGF induced transient phosphorylation of ERK, p38 ( MAPK ) and CREB ... Expression of a dominant negative CREB mutant inhibited Ang II- and EGF dependent induction of the COX-2 promoter
Shanmugam et al., J Mol Cell Cardiol 2011 (Cardiomyopathy, Hypertrophic) : Further, Ang-II also activated CREB via superoxide mediated p38 MAPK and ERK activation
Jin et al., J Biol Chem 2012 : Furthermore, miR-132 up-regulation by Ang II required CREB activation, demonstrating a positive feedback loop