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ICAM1 — SELE

Text-mined interactions from Literome

Knight et al., Transplantation 1999 (Cytomegalovirus Infections) : CMV directly enhanced intercellular adhesion molecule-1 on all EC isolates but did not induce E-selectin or vascular cell adhesion molecule-1
Pasceri et al., Circulation 2000 (Arteriosclerosis...) : Incubation with recombinant human CRP ( 10 microg/mL ) for 24 hours induced an approximately 10-fold increase in expression of ICAM-1 and a significant expression of VCAM-1, whereas a 6-hour incubation induced significant E-selectin expression
Raab et al., Clin Chim Acta 2001 : Activation of endothelial cell monolayers with TNFalpha significantly increases the mean fluorescence intensity of VCAM-1 ( 361+/-14 % ) and ICAM-1 ( 429+/-47 % ) surface expression, compared to controls, and additionally induces E-selectin expression ( 2919+/-134 % )
Ichikawa et al., J Vasc Res 2004 (Anoxia) : Anti-ICAM-1 or anti-P-selectin monoclonal antibodies ( mAbs ) attenuated phase 1 adhesion, while anti-E-selectin mAb attenuated phase 2 adhesion, consistent with roles for constitutive ICAM-1 and preformed P-selectin in early and E-selectin in late phase responses
Enis et al., Proc Natl Acad Sci U S A 2005 (Ischemia) : As in skin, TNF induces E-selectin and vascular cell adhesion molecule 1 only on venular ECs, whereas intercellular adhesion molecule-1 is up-regulated on all human ECs
Latif et al., Cell Biochem Biophys 2005 : Intercellular adhesion molecule-1 was weakly detected , whereas vascular adhesion molecule-1 was barely detectable and E-selectin was not detected
Thornhill et al., J Immunol 1990 : Although, IL-4 stimulation increased 1.4C3 Ag expression ( p less than 0.001 ), it significantly inhibited constitutive ICAM-1 expression ( p less than 0.01 ) and did not induce ELAM-1 ... Furthermore, IL-4 exhibited significant synergy with IL-1 or TNF in inducing 1.4C3 Ag expression ( p less than 0.001 ) but inhibited the increased expression of ICAM-1 produced by IL-1, TNF, or IFN-gamma ( p less than 0.01 ) and inhibited the induction of ELAM-1 by IL-1 and TNF ( p less than 0.001 )
Doukas et al., J Immunol 1990 : IFN-gamma, which by itself increases EC class I MHC and ICAM-1 but does not induce ELAM-1 expression, has been found to act synergistically with TNF to increase class I expression
Damle et al., Eur J Immunol 1991 : Prior activation with phorbol 12-myristate 13-acetate ( PMA ) significantly increased the ability of T cells to up-regulate endothelial ICAM-1 and also induced the expression of both ELAM-1 and VCAM-1
Yoshizaki et al., J Immunol 2010 (Fibrosis...) : This study suggests that L-selectin and ICAM-1 regulate Th2 and Th17 cell accumulation into the skin and lung, leading to the development of fibrosis, and that P-selectin, E-selectin , and PSGL-1 regulate Th1 cell infiltration, resulting in the inhibition of fibrosis
Chiang et al., Prostaglandins Leukot Essent Fatty Acids 2012 : The walnut diet inhibited E-selectin by 12.7 % relative to the fish diet, and the fish diet inhibited secretory intercellular adhesion molecule-1 ( s-ICAM-1 ) by 4.5 % relative to the control diet
Hauser et al., J Immunol 1993 : TNF-alpha increases the expression of ICAM-1 , but not ICAM-2, and induces the expression of ELAM-1 on both EC types
Issekutz et al., J Leukoc Biol 1995 : Antibody blocking studies of the ligands on HUVE indicated that E-selectin may be partially involved in this CD18 independent PMNL migration but that ICAM-1 , VCAM-1, PECAM-1, and P-selectin are not involved
Bryant et al., Infect Immun 1996 : PLC strongly induced expression of ELAM-1 , ICAM-1, and IL-8, while PFO stimulated early ICAM-1 expression but did not promote ELAM-1 expression or IL-8 synthesis ... PLC strongly induced expression of ELAM-1, ICAM-1 , and IL-8, while PFO stimulated early ICAM-1 expression but did not promote ELAM-1 expression or IL-8 synthesis
Zhu et al., Graefes Arch Clin Exp Ophthalmol 1996 (Corneal Diseases...) : In the corneas with BK ( in contrast to normal corneas ), a downregulation of VLA-beta 1 was observed throughout the corneal tissues, particularly on the epithelial layer where bullae occurred ; ICAM-1 was induced on epithelial membranes in both BK and inflamed corneas ; and the expression of beta 2-integrins, VCAM-1 and ELAM-1 was upregulated in some specimens with remaining endothelial cells
Taraboletti et al., Int J Cancer 1997 (Leukemia, Promyelocytic, Acute) : Adhesion was partially mediated by ICAM-1 and, for IL-1 activated endothelium, by VCAM-1 and E-selectin
Shen et al., J Virol 1997 : Expression of E-selectin ( ELAM-1, CD62E ) on human umbilical vein endothelial cells significantly increased 30 min postinfection with the flavivirus West Nile virus ( WNV ), was maximal by 2 h postinfection, and declined to baseline levels within 24 h. Expression of ICAM-1 ( CD54 ) and VCAM-1 ( CD106 ) was significantly increased by 2 h and maximal at 4 h after infection ... TNF-alpha significantly enhanced WNV induced increases in E-selectin , P-selectin, ICAM-1, and VCAM-1 expression, while IFN-gamma enhanced WNV induced ICAM-1 expression
Wittig et al., Exp Cell Res 1997 (Neoplasm Invasiveness) : Soluble E-selectin enhances intercellular adhesion molecule-1 ( ICAM-1 ) expression in human tumor cell lines ... The absence of changes in the expression of vascular cell adhesion molecule, integrin ligands ( CD11a, CD18, integrin alpha 4 ), and sialyl-Lewis X indicates a specific effect of soluble E-selectin on ICAM-1 ... A cell adhesion assay revealed that the enhanced adhesion on T-cells to tumor cells mediated by soluble E-selectin induced ICAM-1 expression was at a maximum after a 12-h incubation period
Coll-Vinent et al., Arthritis Rheum 1998 (Polyarteritis Nodosa) : In early lesions, expression of PECAM-1, ICAM-1 , ICAM-2, and P-selectin was similar to that in control samples, and VCAM-1 and E-selectin were induced in vascular endothelium ... In early lesions, expression of PECAM-1, ICAM-1 , ICAM-2, and P-selectin was similar to that in control samples, and VCAM-1 and E-selectin were induced in vascular endothelium
Lidington et al., Immunology 1998 : PBMC and PBMC supernatants were highly potent at upregulating intercellular adhesion molecule-1 ( ICAM-1 ) and inducing expression of vascular cell adhesion molecule-1 ( VCAM-1 ) and E-selectin
Yamamoto et al., Int Arch Allergy Immunol 1998 : ICAM-1 was enhanced, and E-selectin and VCAM-1 were induced in a dose dependent fashion following stimulation with IL-1beta or TNF-alpha
Nakada et al., Cell adhesion and communication 1998 : VCAM-1 and ICAM-1 cell surface expression were similarly partially inhibited , however, E-selectin levels were unaffected, presumably due to the dual, opposing effect of inhibiting protein expression and inhibiting internalization
Hallahan et al., Cancer Res 1998 : E-selectin expression increased 7-fold, and ICAM-1 expression increased 4-fold after irradiation