Hoare et al., Endocrinology 1999
(Breast Neoplasms) :
We showed the requirement for GABP alpha/beta and c-Fos/c-Jun in endogenous OTR gene expression, using oligonucleotide GABP and AP-1 binding decoys to inhibit serum induced increases in 125I labeled OT antagonist binding to Hs578T cells
Terzidou et al., J Clin Endocrinol Metab 2006
:
In transient transfection studies, OTR promoter activity was increased by C/EBPbeta and NF-kappaB, but not by AP-1