Gene interactions and pathways from curated databases and text-mining

◀ Back to SOD1

ALS7 — SOD1

Text-mined interactions from Literome

Pasinelli et al., Proc Natl Acad Sci U S A 2000 (Amyotrophic Lateral Sclerosis) : Caspase-1 and -3 are sequentially activated in motor neuron death in Cu, Zn superoxide dismutase mediated familial amyotrophic lateral sclerosis
Shimizu et al., J Chromatogr B Analyt Technol Biomed Life Sci 2002 : Variant SOD-1 causes amyotrophic lateral sclerosis
Parton et al., Hum Mutat 2002 (Amyotrophic Lateral Sclerosis) : D90A-SOD1 mediated amyotrophic lateral sclerosis : a single founder for all cases with evidence for a Cis acting disease modifier in the recessive haplotype
Potter et al., J Biol Inorg Chem 2003 (Amyotrophic Lateral Sclerosis) : The perplexing role of copper-zinc superoxide dismutase in amyotrophic lateral sclerosis ( Lou Gehrig 's disease )
Takeuchi et al., J Neurochem 2004 (Amyotrophic Lateral Sclerosis...) : Mutant SOD1s cause familial amyotrophic lateral sclerosis ( FALS ) through the gain of unelucidated toxic properties
Ge et al., J Biol Chem 2005 : The mechanism by which mutated copper-zinc superoxide dismutase ( SOD1 ) causes familial amyotrophic lateral sclerosis is believed to involve an adverse gain of function, independent of the physiological antioxidant enzymatic properties of SOD1
Lobsiger et al., Proc Natl Acad Sci U S A 2005 (Amyotrophic Lateral Sclerosis) : Eliminating assembled neurofilaments ( NFs ) from axons or misaccumulating NFs in motor neuron cell bodies strongly slows disease in mouse models of mutant superoxide dismutase 1 (SOD1) induced amyotrophic lateral sclerosis
Garcia et al., Neurobiol Dis 2006 (Amyotrophic Lateral Sclerosis...) : Mutations in neurofilament genes are not a significant primary cause of non-SOD1 mediated amyotrophic lateral sclerosis
Nawa et al., Cell Signal 2008 (Amyotrophic Lateral Sclerosis) : A novel Akt/PKB interacting protein promotes cell adhesion and inhibits familial amyotrophic lateral sclerosis linked mutant SOD1 induced neuronal death via inhibition of PP2A mediated dephosphorylation of Akt/PKB
Jaarsma et al., J Neurosci 2008 (Amyotrophic Lateral Sclerosis...) : Neuron-specific expression of mutant superoxide dismutase is sufficient to induce amyotrophic lateral sclerosis in transgenic mice
Yang et al., J Neurosci 2009 (Amyotrophic Lateral Sclerosis) : Reticulon-4A ( Nogo-A ) redistributes protein disulfide isomerase to protect mice from SOD1 dependent amyotrophic lateral sclerosis
Graffmo et al., Hum Mol Genet 2013 (Amyotrophic Lateral Sclerosis) : Expression of wild-type human superoxide dismutase-1 in mice causes amyotrophic lateral sclerosis
Zetterström et al., Neuromolecular Med 2013 (Amyotrophic Lateral Sclerosis...) : They may be the primary cause of toxicity in superoxide dismutase-1 induced amyotrophic lateral sclerosis
Saccon et al., Brain 2013 (Amyotrophic Lateral Sclerosis) : Here, we bring together historical and recent experimental findings to conclude that there is a possibility that SOD1 loss of function may play a modifying role in amyotrophic lateral sclerosis
Pickles et al., Hum Mol Genet 2013 (Amyotrophic Lateral Sclerosis...) : Mutant superoxide dismutase 1 (SOD1) selectively associates with spinal cord mitochondria in rodent models of SOD1 mediated amyotrophic lateral sclerosis