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FOS — NPY

Text-mined interactions from Literome

Yokosuka et al., Endocrinology 1999 : Inhibition of neuropeptide Y (NPY) induced feeding and c-Fos response in magnocellular paraventricular nucleus by a NPY receptor antagonist : a site of NPY action
Viñuela et al., J Comp Neurol 2001 : Identification of NPY induced c-Fos expression in hypothalamic neurones projecting to the dorsal vagal complex and the lower thoracic spinal cord ... Temporal analysis of NPY induced c-Fos expression showed a peak at 90 minutes, which was nearly returned to basal levels between 120 and 240 minutes ... In conclusion, NPY induces c-Fos in paraventricular neurones projecting to intermediolateral column of the spinal cord and in neurones of the perifornical nucleus projecting to the dorsal vagal complex
Campbell et al., J Neurosci 2003 : To determine the functional interaction between Y4 receptors and orexin neurons, we examined the effects of rat pancreatic polypeptide ( rPP ), a Y4-selective ligand, or NPY , a nonselective ligand, administered directly into the LHA on the stimulation of food and water intake and c-Fos expression ... Furthermore, both NPY and rPP significantly stimulated c-Fos expression in the LHA ... Neither rPP nor NPY stimulated c-Fos in melanin concentrating hormone neurons, but both activated neurons of an unknown phenotype in the LHA
Brunton et al., Endocrinology 2006 : NPY increased CRH and vasopressin mRNA expression in the parvocellular PVN and stimulated Fos expression in magnocellular supraoptic and PVN oxytocin neurones of virgin but not pregnant rats
Intondi et al., Pain 2008 (Hyperalgesia...) : We found that intrathecal NPY reduced both formalin- and SNI induced Fos expression ... NPY inhibition of SNI induced Fos expression was localized to the sural ( uninjured ) innervation territory, and could be blocked by intrathecal BIBO3304 and BIIE0246
Tsai et al., J Neurotrauma 2009 (Chronic Disease...) : This study sought to investigate the effects of injury induced neuropeptide Y (NPY) on c-Fos expression in the cuneate neurons and neuropathic pain after median nerve injury ... Furthermore, the number of c-Fos-LI cells as well as the percentage of c-Fos-LI cuneothalamic projection neurons (CTNs) in the CN was markedly decreased after injection of NPY receptor antagonist along with retrograde tract tracing method, indicating that NPY regulated c-Fos expression ... Taken together, these findings suggest that more NPY release, following low-intensity electrical stimulation of the injured nerve, significantly induces c-Fos expression in the CTNs, which possibly provide the ascending thalamic transmission of neuropathic pain signals