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SLC2A4 — UBC
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Shibata et al., Rinsho Byori 2005
:
Overexpression of
Ubc9 resulted in inhibition of
GLUT4 degradation and promotion of its targeting to the non-endosomal, non-TGN GLUT4 storage vesicles (GSV), leading to an up-regulation of GLUT4 expression level and insulin-responsive glucose transport
Liu et al., Diabetes 2007
:
Overexpression of
Ubc9 resulted in an inhibition of GLUT4 degradation and
promoted its targeting to the unique insulin-responsive
GLUT4 storage compartment (GSC) , leading to an increase in GLUT4 amount and insulin-responsive glucose transport in 3T3-L1 adipocytes