◀ Back to SLC22A3

GPI — SLC22A3

Text-mined interactions from Literome

Funasaka et al., Cancer Res 2009 (Breast Neoplasms) : Taken together, the results show the involvement of PGI/AMF in both EMT and MET : overexpression of PGI/AMF induces EMT in normal breast epithelial cells and reduction of PGI/AMF expression led to MET in aggressive breast cancer cells
Ahmad et al., Cancer Res 2011 (Breast Neoplasms...) : We have previously shown its role in the induction of epithelial-mesenchymal transition (EMT) in breast cancer cells, which led to increased aggressiveness ; however, the molecular mechanism by which PGI/AMF regulates EMT is not known ... Collectively, these results suggest a role of miR-200s in PGI/AMF induced EMT and thus approaches for upregulation of miR-200s could be a novel therapeutic strategy for the treatment of highly invasive breast cancer