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GCG — NOS1

Text-mined interactions from Literome

Aring;kesson et al., J Physiol 1999 : 4. In islets depolarized with 30 mM K+ in the presence of the KATP channel opener diazoxide, NOS inhibition by 5 mM L-NAME still markedly potentiated carbachol induced insulin release ( although less so than in normal islets ) and suppressed glucagon release
Ding et al., Am J Physiol Endocrinol Metab 2000 (Hyperglycemia) : Effects of simulated hyperglycemia, insulin, and glucagon on endothelial nitric oxide synthase expression
Akesson et al., Br J Pharmacol 1996 : 2. In freely fed mice, L-NAME pretreatment ( 1.2 mmol kg-1 ) influenced the dynamics of insulin and glucagon release following an equimolar dose of L-arginine, the specific substrate for NOS activity, in that the NOS inhibitor enhanced the insulin response but suppressed the glucagon responses
Akesson et al., Biosci Rep 1998 : It was observed that the NO synthase (NOS) inhibitor N ( G ) -nitro-L-arginine methylester ( L-NAME ) markedly potentiated insulin release and modestly inhibited glucagon release induced by carbachol