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BAX — EGFR

Text-mined interactions from Literome

Wang et al., Zhonghua Zhong Liu Za Zhi 2003 (Glioblastoma) : Bcl-2 and Bax , being nuclear matrix associated proteins, are probably involved in the EGFR-cDNA induced malignant conversion of glioblastoma cells by introducing EGFR cDNA into the tumor cells
Bernal et al., Gastroenterology 2006 : Epidermal growth factor receptor signaling regulates Bax and Bcl-w expression and apoptotic responses during intestinal adaptation in mice
Ge et al., Biol Reprod 2009 : On the other hand, inhibition of EGFR , Ca ( 2+ ) /PKC, or NFKB1 abolished the EGF stimulated increase in the expression of cyclins CCND1 and CCNE1, cyclin dependent kinase 6 (CDK6), CDK2, and BCL2, and restored the EGF induced inhibition of BAX expression and caspase 3/9 activity, indicating that EGFR, PKC, and NFKB1 signaling cascades were involved in EGF stimulated DNA synthesis and antiapoptosis action
Yan et al., Gastroenterology 2009 (Disease Models, Animal...) : Furthermore, H pylori induced EGFR phosphorylation stimulated phosphotidylinositol-3'-kinase dependent activation of the antiapoptotic factor Akt, increased expression of the antiapoptotic factor Bcl-2, and decreased expression of the proapoptotic factor Bax