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HDAC3 — MYLIP

Text-mined interactions from Literome

Zhou et al., PloS one 2012 (MAP Kinase Signaling System) : Importantly, miR-16 targeted the 3'-untranslated region of SMRT and caused translational suppression of SMRT
Zhang et al., Cancer Cell 2012 (Lymphoma, B-Cell) : We investigated the transcriptional and epigenetic repression of miR-29 by MYC, HDAC3 , and EZH2 in mantle cell lymphoma and other MYC associated lymphomas
Li et al., Blood 2013 (Leukemia, Myeloid, Acute) : Suppression of miR-193a expands the oncogenic activity of the fusion protein AML-ETO, because miR-193a represses the expression of multiple target genes, such as AML1/ETO, DNMT3a, HDAC3 , KIT, CCND1, and MDM2 directly, and increases PTEN indirectly
Lodrini et al., Nucleic Acids Res 2013 (Neuroblastoma) : In profiling studies, histone deacetylase (HDAC) inhibitor treatment most strongly induced miR-183