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GP9 — IL10
Text-mined interactions from Literome
Wessells et al., Eur J Immunol 2000
:
Pregnancy specific
glycoprotein 18
induces IL-10 expression in murine macrophages
Pukhal'skii et al., Bull Exp Biol Med 2001
(Inflammation) :
We studied the
effects of alpha1-acid
glycoprotein on tumor necrosis factor-alpha (TNF-alpha) and
interleukin-10 (IL-10) production and lymphocyte response to phytohemagglutinin in cultured peripheral blood mononuclear leukocytes from 6 healthy donors
Ha et al., J Leukoc Biol 2005
:
Binding of pregnancy-specific
glycoprotein 17 to CD9 on macrophages
induces secretion of
IL-10 , IL-6, PGE2, and TGF-beta1
Sundar et al., Proc Natl Acad Sci U S A 1991
:
Human immunodeficiency virus
glycoprotein ( gp120 ) infused into rat brain
induces interleukin 1 to elevate pituitary-adrenal activity and decrease peripheral cellular immune responses
Lee et al., Mol Cell Biochem 2013
(Carcinoma, Hepatocellular...) :
The results revealed that the SJSZ
glycoprotein ( 10 mg/kg, BW )
inhibits the phosphorylation of MAPKs and expression of NF-?B, pSTAT6,
IL-10 , and IL-4 ; and normalizes production of ROS and NO, and expression of iNOS, IL-12, and IFN-?
Perona-Wright et al., Proc Natl Acad Sci U S A 2012
(Respiratory Tract Infections) :
CD8+ memory T cells secreted less
IL-10 when activated in the presence of IL-27 than did naïve controls, and retroviral expression of
glycoprotein 130
restored IL-10 and reduced IFN-? production upon restimulation
Lee et al., Inflammation 2013
:
The SJSZ
glycoprotein normalized production of NO and ROS and expression of inducible nitric oxide synthase, IFN-?, and IL-12 but
suppressed expression of pSTAT6, IL-4,
IL-10 , and CD163 positive cells
Kojima et al., J Pharmacobiodyn 1986
(Neoplasms, Experimental) :
Furthermore, this serum
glycoprotein was found to
enhance glucose consumption and
interleukin-1 production and cytotoxic activities of mouse peritoneal macrophages
Geiger et al., J Biol Chem 1988
(Liver Neoplasms, Experimental) :
Induction of alpha 1-acid
glycoprotein by recombinant human
interleukin-1 in rat hepatoma cells
Weston et al., Transpl Immunol 1995
(Kidney Failure, Chronic) :
E-Selectin is a 115-kDa cell surface
glycoprotein transiently expressed on vascular endothelium in
response to
interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Fournier et al., Hepatology 1994
:
These results show that ( a ) phenobarbital acts directly on hepatocytes by increasing alpha 1-acid glycoprotein gene expression and ( b ) this effect is mediated by a specific mechanism independent of pathways involved in alpha 1-acid
glycoprotein induction by
interleukin-1 , interleukin-6 and glucocorticoids
Herbert et al., FEBS Lett 1993
:
Endotoxin ( LPS ),
interleukin-1 beta (IL-1) and tumor necrosis factor-alpha (TNF)
increased the expression of tissue factor, a membrane anchored
glycoprotein that initiates blood coagulation on the surface of cultured bovine aortic endothelial cells ( ABAE ) and human monocytes
Andrejko et al., Crit Care Med 1996
(Sepsis) :
To test the hypothesis that after cecal ligation and puncture in the rat, there is increased expression of the tumor necrosis factor
(TNF)/interleukin-1 dependent , acute-phase reactant alpha 1-acid
glycoprotein in the liver, and that this change correlates temporally with increased abundance of TNF-alpha in the hepatic parenchyma but not with circulating concentrations of TNF-alpha