We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to BCL10

BCL10 — PI3

Text-mined interactions from Literome

Leverrier et al., Cell Death Differ 1999 : Role of PI3-kinase in Bcl-X induction and apoptosis inhibition mediated by IL-3 or IGF-1 in Baf-3 cells ... In this study, we have investigated the role of PI3-kinase in the regulation of Bcl-X expression and in the survival of Baf-3 cells
Liu et al., Oncogene 2000 (MAP Kinase Signaling System) : The PI-3K specific inhibitor wortmannin did not affect hGM-CSF dependent anti gamma irradiation induced apoptosis nor bcl-X ( L ) induction, thus bcl-X ( L ) but not PI-3K pathway seems to be involved in hGM-CSF dependent anti gamma irradiation induced apoptosis
Fabregat et al., Hepatology 2000 : PI 3-kinase inhibitors completely block the protective effect of EGF on TGF-beta induced bcl-x ( L ) down-regulation
Fornoni et al., Am J Pathol 2001 : The regulation of Bcl-xL by HGF was mediated by the PI3'-K but not by the MEK-1 pathway
van der Kuip et al., Blood 2001 : The fibronectin mediated antiapoptotic mechanism in Bcr-Abl-active cells was not mediated by overexpression of Bcl-XL or Bcl-2 but required an active phosphatidylinositol 3-kinase (PI-3K)
Bae et al., Cancer Res 2006 (Adenocarcinoma...) : Bcl-w overexpression also activated phosphoinositide 3-kinase (PI3K) , Akt, and Sp1, and the blocking effects of each of these components using pharmacologic inhibitors, dominant negative mutants, or small interfering RNA abolished the ability of Bcl-w to induce MMP-2 and cell invasion
Moriya et al., Hepatology 2012 (Disease Models, Animal...) : This retained expression of Bcl-xL is inhibited by the phosphatidylinositol 3-kinase (PI3K) inhibitor LY294002