◀ Back to IL8

IL8 — TNFSF13B

Text-mined interactions from Literome

Scapini et al., Blood 2005 (Arthritis, Rheumatoid) : We show that CXCL1/GROalpha, CXCL8/IL-8 , C5a, immune complexes, tumor necrosis factor-alpha (TNF-alpha), leukotriene B4, N-formyl-methionyl-leucyl-phenylalanine ( fMLP ), and lipopolysaccharide (LPS), which by themselves do not induce BLyS de novo synthesis, act as potent secretagogues for BLyS, which is mainly stored in Golgi related compartments within G-CSF treated neutrophils, as determined by immunogold electron microscopy
Jeon et al., Immunol Cell Biol 2010 : The stimulation of BAFF induced the expression of matrix metalloproteinase (MMP)-9, interleukin -8 , and transforming growth factor-beta induced gene product ( beta ig-h3 ) and the upregulation of intercellular adhesion molecule-1 in THP-1 cells
Lee et al., Immunology 2011 : Synthetic peptides containing ITIM-like sequences of IREM-1 inhibit BAFF mediated regulation of interleukin-8 expression and phagocytosis through SHP-1 and/or PI3K ... Interestingly, all five of these synthetic peptides, representing the five ITIM-like sequences present in the cytoplasmic tail of IREM-1, exhibited inhibitory action against BAFF mediated induction of matrix metalloproteinase-9 and interleukin-8 expression
Yasuda et al., Food Funct 2013 : In a co-culture system consisting of differentiated Caco-2 cells and RAW264.7 macrophages, mRNA expression of IL-6, IL-8, IL-1ß and BAFF cytokines was up-regulated in Caco-2 cells and IL-8 production in basolateral medium was induced after 24 h apical treatment with 5 mg ml(-1) of ESG