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CSF2 — MYC

Text-mined interactions from Literome

Cheng et al., J Biol Chem 1999 : Using the mitogen activated protein kinase kinase ( MEK1/2 ) inhibitor PD98059 and dominant negative forms of Ras ( N17 ) and ERK1 ( K71R ), we found that activation of Ras and extracellular signal regulated kinase ( ERK ) is necessary for colony stimulating factor-1 (CSF-1) mediated c-Myc expression and DNA synthetic ( S ) phase entry
Kobayashi et al., Blood 2003 (Leukemia) : To investigate the roles of c-myc during hematopoietic proliferation induced by growth factors, we used factor dependent human leukemic cell lines ( MO7e and F36P ) in which proliferation, cell cycle progression, and c-Myc expression were strictly regulated by granulocyte-macrophage colony stimulating factor ( GM-CSF ) and interleukin-3 (IL-3) ... In these cell lines, both c-myc mRNA and c-Myc protein stability were not affected by GM-CSF and IL-3, suggesting a regulation of c-Myc protein at the translational level
Aziz et al., Mol Cell Biol 1999 : Activation of the human colony stimulating factor 1 (CSF-1) receptor in NIH 3T3 cells leads to activation of the mitogen activated protein ( MAP ) kinase pathway and induced expression of c-Fos, c-Myc , and cyclin D1, leading to a potent mitogenic response