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MMP9 — MYD88

Text-mined interactions from Literome

Gorina et al., Glia 2011 : p38 exerted a strong influence on LPS induced gene expression by regulating the phosphorylation of Stat1 and the transcriptional activity of NF?B, while JNK regulated the Jak1/Stat1 pathway, and ERK1/2 controlled the expression of Egr-1 and influenced MyD88 dependent MMP-9 expression
Wang et al., Cancer Biol Ther 2012 (Lung Neoplasms) : We observed a MyD88 dependent upregulation of matrix metalloproteinase (MMP) 2, MMP9 and cyclin dependent kinase (CDK) 2 in 95D cells in response to HMGB1
Abdulkhalek et al., Cell Signal 2013 (Inflammation) : Tamiflu, specific MMP-9 inhibitor, neuromedin B receptor specific antagonist BIM23127, and the selective inhibitor of whole heterotrimeric G-protein complex BIM-46174 significantly block nucleic acid induced TLR-7 and -9 MyD88 recruitment, NF-?B activation and proinflammatory TNFa and MCP-1 cytokine responses