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AGO2 — PTGS2

Text-mined interactions from Literome

Boutet et al., Curr Biol 2003 : Here, we show that : i ) SGS2, SGS3, AGO1, and HEN1 contribute to produce transgene siRNA involved in sense posttranscriptional gene silencing ( S-PTGS ) ; ii ) HEN1, but not SGS2, SGS3, or AGO1, contributes to the accumulation of the endogenous miR171 miRNA and to the cleavage of Scarecrow target mRNA by miR171 ; iii ) SGS2, SGS3, AGO1 , and HEN1 contribute to resistance against cucumber mosaic virus, but not to siRNA and IR-PTGS triggered by hairpin transgenes directly producing perfect dsRNA ; and iv ) the actions of HEN1 in miRNA/development and siRNA/S-PTGS can be uncoupled by single-point mutations at different positions in the protein
Huang et al., Toxicol Lett 2007 (Cell Transformation, Viral) : In addition, our immunocytochemical and Western blotting results showed that p-PD induced overexpression of mutant p53 and COX-2 in a dose dependent manner ... These results suggest that p-PD can induce DNA damage and accumulation of mutant p53 and COX-2 proteins ; this may be one of the possible mechanisms that cause genotoxic carcinogenesis in the urothelial cells
Duan et al., Plant Cell 2012 : Furthermore, the direct 2b-AGO interaction was not essential for the 2b suppression of posttranscriptional gene silencing ( PTGS ) and RNA directed DNA methylation ( RdDM ) in vivo