Yang et al., Immunity 2008
(Encephalomyelitis, Autoimmune, Experimental...) :
Although TGF-beta receptor I signaling was required for both Foxp3 and IL-17 induction, SMAD4 was only involved in Foxp3 upregulation
Schlenner et al., J Exp Med 2012
(Colitis) :
In vitro, Smad signaling facilitates conversion of naive T cells into T reg cells but results in unstable Foxp3 expression