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Text-mined interactions from Literome

Yamashita et al., J Biol Chem 2001 : The results support essential roles for AP-1 , GATA-2, and NF-1 in stabilizing the binding of HIF-1 and promoting recruitment of p300/CBP to the ET-1 hypoxia response complex
Tacchini et al., Carcinogenesis 2001 (Carcinoma, Hepatocellular...) : Third, consistent with these data, N-acetylcysteine reduced the stimulatory effect of HGF on stress kinase activities, while p42/44 mitogen activated kinase ( MAPK ) was unmodified, suggesting an involvement of c-Jun-N-terminal kinase (JNK) and p38 MAPK in HIF-1 activation
Laderoute et al., Mol Cell Biol 2004 (MAP Kinase Signaling System) : Here we describe new findings concerning HIF-1 dependent c-Jun N-terminal phosphorylation in cells exposed to hypoxia or anoxia
Nanduri et al., Respir Physiol Neurobiol 2008 (Anoxia) : Compared to same duration and intensity of CH, IH is more potent in activating HIF-1 and c-fos and also results in long lasting accumulation of HIF-1alpha and c-fos mRNA, a phenomenon that was not seen with CH. IH-evoked transcriptional activation by HIF-1, c-fos as well as the resulting activator protein-1 (AP-1) requires reactive oxygen species ( ROS ) -mediated signaling and involves complex feed forward interactions between HIF-1 and ROS