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AKT3 — PLCG1

Text-mined interactions from Literome

Hong et al., Biochem Biophys Res Commun 2001 : In addition, PLC-gamma1 activation was independent of Akt/protein kinase B ( Akt/PKB )
Kwon et al., J Cell Biol 2003 : Expression of the PH-EF construct blocked endogenous PLCgamma1 binding to the IR and inhibited insulin dependent phosphorylation of mitogen activated protein kinase ( MAPK ), but not AKT
Nguyen et al., J Biochem Mol Biol 2007 (MAP Kinase Signaling System) : By contrast, Overexpression of WT-PLC-gamma1 exhibited sustained NGF induced MAPK activation, and triggered transient Akt activation resulting in profound inhibition of neurite outgrowth
Zheng et al., J Neurochem 2008 (Ion Channel Gating) : Although previous studies suggested the roles of mitogen activated protein kinase, phospholipase C-gamma mediated intracellular calcium ( [ Ca2+ ] i ) increase, and extracellular calcium influx in regulating Akt activation, the cellular mechanisms are largely unknown
Maffucci et al., PloS one 2009 : Finally we show that PI3K dependent PLCgamma1 activation regulates FGF-2 mediated phosphorylation of Akt at its residue Ser473, determined by Western blotting analysis
Coso et al., PloS one 2012 : Furthermore, activation of PI3K/Akt by VEGF-C/VEGFR-3 resulted in phosphorylation of P70S6K, eNOS, PLC?1 , and Erk1/2