◀ Back to NGF

NGF — PDCD1

Text-mined interactions from Literome

Frade et al., Microsc Res Tech 1999 : Thus, NT-3 promotes the conversion of neuroepithelial cells into neurons, whereas BDNF and NGF control the programmed cell death ( apoptosis ) that affects early postmitotic neuroblasts
Mayumi-Matsuda et al., Biochim Biophys Acta 1999 : Depletion of nerve growth factor (NGF) from differentiated, neuronal PC12 cells causes a form of programmed cell death that stems from the attenuation of NGF receptor signaling and the resultant expression of certain genes required for cell death
Gentry et al., J Biol Chem 2000 (Neurilemmoma) : In contrast to its trophic actions through TrkA, NGF binding to p75 has been shown to activate programmed cell death through a mechanism involving the stress kinase JNK
Deshmukh et al., J Cell Biol 2000 : Nerve growth factor (NGF) deprivation induces a Bax dependent, caspase dependent programmed cell death in sympathetic neurons
Niederhauser et al., J Neurosci Res 2000 : In SK-N-MC cells this leads to inhibition of NGF induced programmed cell death
Peltier et al., Curr Opin Neurol 2002 (Peripheral Nervous System Diseases) : In contrast, nerve growth factor prevents initiation of the programmed cell death associated with cisplatin neurotoxicity
Jelaso et al., Int J Dev Neurosci 2005 : NGF , a neurotrophic factor best known for its role in promoting cell survival, regulates many neurodevelopmental processes, including synaptic plasticity, neurite outgrowth and programmed cell death
Ito et al., J Neurochem 1995 : Ceramide prevents neuronal programmed cell death induced by nerve growth factor deprivation
Mills et al., J Neurochem 1996 : Terminally differentiated PC12 cells are a useful neuron-like model for studying programmed cell death in response to nerve growth factor (NGF) deprivation
Frade et al., Development 1999 : The role of nerve growth factor (NGF) and of the neurotrophin receptor p75 ( p75 ( NTR ) ) in programmed cell death was investigated in the retina and the spinal cord of mouse embryos
Sanz et al., Oncogene 1999 : The increase in c-Raf levels also prevented nerve growth factor (NGF) dependent induction of programmed cell death