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EGFR — NUP43
Text-mined interactions from Literome
Xie et al., Eur J Biochem 2002
:
The signaling pathways that are rapidly elicited by the interaction of ouabain with Na ( + ) /K ( + ) -ATPase, and are independent of changes in intracellular Na ( + ) and K ( + ) concentrations, include activation of Src kinase, transactivation of the
epidermal growth factor receptor by Src,
activation of Ras and
p42/44 mitogen activated protein kinases, and increased generation of reactive oxygen species by mitochondria
Li et al., J Biol Chem 2005
:
Tyrosine phosphorylation of Srcasm is dependent on growth factors and the activity of
EGFR and SFKs. Increased Srcasm expression
enhances p44/42 mitogen activated protein kinase activity and Elk-1 dependent transcriptional events
Nkabyo et al., Am J Physiol Gastrointest Liver Physiol 2005
(MAP Kinase Signaling System) :
Inhibitors of
EGFR ( AG1478 ) and p44/p42 MAPK ( U0126 ) phosphorylation
blocked redox dependent
p44/p42 phosphorylation, indicating that signaling occurred by EGFR
Lee et al., American journal of physiology. Renal physiology 2008
:
In addition, the PKC inhibitors or an
EGFR inhibitor ( AG-1478 )
blocked the BSA induced phosphorylation of
p44/42 mitogen activated protein kinases ( MAPKs )