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CA2 — KCNMA1
Text-mined interactions from Literome
Yamamura et al., Br J Pharmacol 2001
:
BK channel activation by NS-1619 is partially
mediated by intracellular
Ca2+ release in smooth muscle cells of porcine coronary artery
Chung et al., Expert Opin Investig Drugs 2002
(Cough) :
Local anaesthetics, blockers of sodium dependent channels and
maxi-K Ca2+ dependent channel activators of afferent nerves are inhibitors of the cough reflex
Chung et al., Pulm Pharmacol Ther 2002
(Cough) :
Local anaesthetics, blockers of sodium dependent channels, and
maxi-K CA2+ dependent channel activators of afferent nerves are inhibitors of the cough reflex
Liu et al., EMBO J 2004
:
Assembly of a
Ca2+ dependent
BK channel signaling complex by binding to beta2 adrenergic receptor
Lu et al., J Physiol 2006
(Calcium Signaling...) :
As
MaxiK senses and
regulates membrane voltage and intracellular
Ca2+ , it links cell excitability with cell signalling and metabolism
Misonou et al., J Comp Neurol 2006
:
Immunolocalization of the
Ca2+ activated K+ channel
Slo1 in axons and nerve terminals of mammalian brain and cultured neurons
Chiang et al., J Exp Biol 2006
:
In the Rhabditidae family, although M4 normally has no effect on the terminal bulb, we found that M4 can stimulate the terminal bulb in C. elegans if the
Ca2+ activated K+ channel
SLO-1 is inactivated
Latorre et al., Biol Res 2006
(Ion Channel Gating) :
The
BK channel is
activated by internal
Ca2+ , and using chimeric channels and mutagenesis, three distinct Ca2+ dependent regulatory mechanisms with different divalent cation selectivity have been identified in its large COOH-terminus
Kaufmann et al., J Comp Neurol 2009
:
Such increases in cytosolic
[Ca2+ ] may be
sufficient for
BK channel activation
Latorre et al., J Physiol 2010
:
Allosteric interactions and the modular nature of the voltage- and
Ca2+ activated (
BK) channel
Guggino et al., Am J Physiol 1987
:
We conclude that cultures of MTAL cells GRB-MAL1 retain at least two of the properties of the mature phenotype, namely, an apical K+ conductance and a sensitivity to loop diuretics ; the most frequently occurring channel in the apical cell membrane is a
Ca2+ activated ,
maxi-K+ channel ; and, finally Ca2+ activated K+ channels may play a role in generating the apical K+ conductance in cultured MTAL cells
Taniguchi et al., Am J Physiol 1989
:
The effects of membrane stretch on
Ca2+ activated (
maxi) K+ channels were examined in the apical membrane of cultured medullary thick ascending limb ( MTAL ) cells
Romero et al., Am J Physiol 1998
:
Evidences that activation of the
maxi-K+ channel by ANG II
requires a rise in intracellular
Ca2+ concentration ([Ca2+]i) as an intermediate step were the shift of the open probability of the channel-membrane potential relationship to less positive membrane potentials and the sustained increase in [ Ca2+ ] i in fura 2-loaded myocytes