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ATP5O — PI3

Text-mined interactions from Literome

Alexander et al., Am J Physiol Gastrointest Liver Physiol 2001 : Involvement of PI 3-kinase in IGF-I stimulation of jejunal Na+-K+-ATPase activity and nutrient absorption
Muscella et al., J Cell Physiol 2005 (Breast Neoplasms) : Additionally, the effect of Ang II on Na+/K+ATPase activity was also blocked by the phosphatidylinositol 3-kinase (PI3K) inhibitors, wortmannin and LY294002, and by the actin depolymerizing agents, cytochalasin D
Garofalo et al., Proc Biol Sci 2009 : Using the same bioassay, we now demonstrate that this effect is confirmed in the presence of the exogenous NO donor S-nitroso-N-acetyl penicillamine, is independent from endocardial endothelium and guanylate cyclase/cGMP/protein kinase G and cAMP/protein kinase A pathways, involves a PI(3)kinase mediated activation of endothelial NO synthase and a modulation of the SR-CA(2+)ATPase ( SERCA2a ) pumps
Lei et al., Am J Physiol Lung Cell Mol Physiol 2011 : Inhibitors of Src kinase ( PP1 ), PI3K ( wortmannin ), and ERK1/2 ( U0126 ) all blocked the T3-induced Na-K-ATPase activity