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CDKN1A — HDAC7

Text-mined interactions from Literome

Xiao et al., J Cell Biochem 1999 : Both Sp1 and Sp3 are responsible for p21waf1 promoter activity induced by histone deacetylase inhibitor in NIH3T3 cells
Sowa et al., Cancer Res 1999 : Sp3, but not Sp1, mediates the transcriptional activation of the p21/WAF1/Cip1 gene promoter by histone deacetylase inhibitor
Xiao et al., J Biol Chem 2000 : p300 collaborates with Sp1 and Sp3 in p21 ( waf1/cip1 ) promoter activation induced by histone deacetylase inhibitor
Shin et al., Cancer Res 2000 (Stomach Neoplasms) : The p21 gene was activated only by histone deacetylase inhibitor
Claassen et al., Proc Natl Acad Sci U S A 2000 : Repression of p21 ( CIP1 ) transcription by c-Myc occurred at the promoter level in a region near the start site of transcriptional initiation and was independent of histone deacetylase activity
Huang et al., Oncogene 2000 (Breast Neoplasms) : Activation of the p21WAF1/CIP1 promoter independent of p53 by the histone deacetylase inhibitor suberoylanilide hydroxamic acid ( SAHA ) through the Sp1 sites
Sowa et al., Biofactors 2000 (Colorectal Neoplasms...) : As a model of this, we found that histone deacetylase inhibitors such as butyrate or trichostatin A dramatically stimulate the p21/WAF1 gene promoter through the Spl sites, resulting in cell cycle arrest
Han et al., J Biol Chem 2001 : Activation of p21 ( WAF1/Cip1 ) transcription through Sp1 sites by histone deacetylase inhibitor apicidin : involvement of protein kinase C
Burgess et al., Mol Pharmacol 2001 : Up-regulation of p21 ( WAF1/CIP1 ) by histone deacetylase inhibitors reduces their cytotoxicity
Blagosklonny et al., Mol Cancer Ther 2002 : By preventing deacetylation of histones, histone deacetylase inhibitors ( HDIs ) transcriptionally induce p21
Sowa et al., Nihon Eiseigaku Zasshi 2003 (Neoplasms) : As a model of our hypothesis, we found that histone deacetylase inhibitors such as butyrate and trichostatin A dramatically stimulate the p21/WAF1 gene promoter through the Sp1 sites, resulting in cell cycle arrest
Piekarz et al., Blood 2004 (Lymphoma, T-Cell) : Treatment with depsipeptide, as well as other histone deacetylase inhibitors, caused induction of histone acetylation, induction of p21 expression, and substantial apoptosis without significant cell cycle arrest
Malinen et al., Nucleic Acids Res 2008 (Breast Neoplasms) : HDAC3 and HDAC7 attenuated 1alpha,25 ( OH ) 2D3 dependent induction of the p21 gene, for which NCoR1 is essential