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HNRNPH1 — RELA

Text-mined interactions from Literome

Ruby et al., Mol Pharmacol 2002 : Dendritic cells (DCs) , considered to be the `` pacemakers '' of the immune system, were recently recognized as targets of TCDD and are also dependent on NF-kappaB/Rel for activation and survival
Hu et al., Infect Immun 2006 : In addition, Campylobacter infected DCs triggered activation of NF-kappaB and significantly stimulated production of interleukin-1beta (IL-1beta), IL-6, IL-8, IL-10, IL-12, gamma interferon, and tumor necrosis factor alpha (TNF-alpha) compared to uninfected DCs
Golks et al., J Exp Med 2006 (Neoplasms) : Here, we demonstrate that the cytoplasmic NH2-terminal procaspase-8 cleavage product of c-FLIP ( p22-FLIP ) found in nonapoptotic malignant cells, primary T and B cells, and mature dendritic cells (DCs) strongly induces nuclear factor kappaB (NF-kappaB) activity by interacting with the IkappaB kinase (IKK) complex via the IKKgamma subunit
Xu et al., J Biol Chem 2009 (Calcium Signaling) : In addition, PLCgamma2-deficient DCs are also impaired in their activation of NF-kappaB upon Dectin-1 engagement due to defective assembly of the Card9-Bcl10-Malt1 complex and impaired IKKalpha/beta activation and IkappaBalpha degradation
Zhu et al., J Interferon Cytokine Res 2009 : DCs treated with HMGB1 ( 1 microg/mL for 48 h ) promoted T-cell proliferation as well as differentiation, and markedly up-regulated IL-2, IL-2R expression and intranuclear NF-kappaB activation
Bansal et al., J Immunol 2010 (Tuberculosis) : We further found that PE_PGRS protein mediated activation of DCs involves participation of ERK1/2, p38 MAPK, and NF-kappaB signaling pathways
Yekollu et al., Diabetes 2011 (Insulin Resistance...) : Targeting Tip-DCs and ATMs with curcusomes in ob/ob mice reduced NF-?B/RelA DNA binding activity, reduced TNF, and enhanced interleukin-4 production