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FAS — PCBD1

Text-mined interactions from Literome

Myc et al., Endocrinology 1999 : In addition, in the presence of a suboptimal concentration of CHX, the Ac-SLV tripeptide yielded a strong, synergistic increase in Fas mediated PCD as compared to thyrocytes treated with control tripeptide ... These results implicate FAP-1 as a regulator of Fas induced PCD in thyrocytes
Raoul et al., J Cell Biol 1999 : In the presence of neurotrophic factors, exogenous Fas activators such as soluble FasL or anti-Fas antibodies triggered PCD of 40-50 % of purified motoneurons over the following 3-5 d ; this treatment led to activation of caspase-3, and was blocked by IETD
Ramenghi et al., Blood 2000 (Autoimmune Diseases...) : Fas/Apo-1 ( CD95 ) triggers programmed cell death (PCD) and is involved in immune response control and cell mediated cytotoxicity
Zazzeroni et al., Blood 2003 : In B lymphocytes, induction of apoptosis or programmed cell death (PCD) by Fas ( CD95/APO-1 ) is suppressed by the triggering of CD40
Kontny et al., Adv Exp Med Biol 2006 (Arthritis, Rheumatoid) : In leukemic Jurkat cells Tau-Cl ( > 200 microM ) triggers mitochondrial, p53 independent apoptosis and amplifies PCD induced by anti-Fas treatment
Van Parijs et al., Immunity 1996 : This form of programmed cell death (PCD) is not dependent on Fas , and is prevented by CD28 mediated signals, which lead to the secretion of growth factors and the expression of survival genes, such as bcl-xL