◀ Back to EPHB2

DEFA1B — EPHB2

Text-mined interactions from Literome

Aarbiou et al., Am J Respir Cell Mol Biol 2004 (MAP Kinase Signaling System) : Furthermore, HNP1-3 caused a biphasic activation of the mitogen activated protein kinase extracellular regulated kinase 1 and 2 ( ERK1/2 ) ... Both the effects of HNP1-3 on wound closure and ERK1/2 activation were blocked by specific inhibitors of the mitogen activated protein kinase kinase MEK, whereas inhibitors of epidermal growth factor receptor tyrosine kinase, phosphatidylinositol 3-kinase, and Src did block defensin enhanced wound closure but not ERK1/2 activation
Zuyderduyn et al., J Allergy Clin Immunol 2006 (Inflammation) : HNP1-3 did increase ERK1/2 phosphorylation, but did not enhance IL-8 release by HASM cells