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BCL10 — PARP8

Text-mined interactions from Literome

Kim et al., J Neurochem 1999 : The sequential cleavage of PARP and the appearance of the small Bax-immunoreactive protein in MN9D cells were blocked either by Z-VAD-fmk or by Bcl-X ( L )
Jang et al., Biochem Biophys Res Commun 2002 : The L-canavanine induced caspase-3 activation, degradation of PARP , and apoptotic DNA fragmentation were suppressed by ectopic expression of Bcl-2 or Bcl-xL , both of which are known to play roles as anti-apoptotic regulators
Kawano et al., Anticancer Res 2004 (MAP Kinase Signaling System) : The combination treatment activated caspase-3 and cleaved PARP , but it did not induce any notable change in the expression of Bcl-XL , Bcl-2 and Bax compared with each single treatment
Ibrado et al., Cancer Res 1996 : In the present studies, we determined the effect of high intracellular levels of the antiapoptosis Bcl-2 or Bcl-xL protein on Yama protease activation and PARP degradation during Ara-C induced apoptosis ... High Bcl-2 and Bcl-xL levels in these cells also inhibited Yama protease activity, PARP degradation, and apoptosis due to clinically relevant concentrations of etoposide and mitoxantrone